![]() At The Johns Hopkins Hospital, we have diagnosed SSAs since 2001 based on awareness of work by Torlakavik and Snover. They are less common than conventional colorectal adenomas and were largely undiagnosed in general pathology and gastroenterology practice until about 2005. Sessile serrated adenomas (SSAs) are colonic polyps with a serrated glandular pattern and architectural features that overlap with those of hyperplastic polyps. These results suggest that guidelines for following up patients with sessile serrated adenomas as per advanced adenomas are warranted. Those with follow-up were managed as per advanced adenomas their clinical outcomes supported this. The follow-up of sessile serrated adenomas from the study period (2002 to 2004) was more rigorous than proposed for sporadic tubular adenomas (patients with sporadic tubular adenomas were also followed up more aggressively than suggested by guidelines). Most (53/66, 80.3%) patients had tubular adenomas on follow-up, 12 (18.2%) of 66 patients had hyperplastic polyps, and 2 (3.0%) of 66 patients had a sessile serrated adenoma. Sixty-six patients (71.7%) received follow-up colonoscopy. Ninety-two patients with tubular adenomas between January 2002 and December 2004 formed the control group. Sessile serrated adenomas were found in 22 (51.2%) of 43 patients, 16 (37.2%) of 43 patients had tubular adenomas, and hyperplastic polyps were diagnosed in 18 (41.9%) of 43. Mucinous adenocarcinoma was diagnosed in 1 (2.3%) of 43 patients, and 1 (2.3%) of 43 patients had high-grade dysplasia in an sessile serrated adenoma. One or more lesions were found in 42 (97.6%) of 43 patients. Forty three patients (46.2%) had follow-up colonoscopy. Ninety-nine sessile serrated adenomas from 93 patients were diagnosed between January 2002 and December 2004. A control group of patients with sporadic tubular adenomas was selected. Materials from patients diagnosed with sessile serrated adenoma from January 2002 to December 2004 were reviewed. We evaluated follow-up of patients with sessile serrated adenoma diagnosed between 20 in our teaching institution and compared it to follow-up of randomly selected tubular adenomas. ![]() Your health care provider might recommend stopping proton pump inhibitors or removing the polyp or both.Sessile serrated adenomas (SSAs) were unrecognized in pathology and gastroenterology practice until about 2005 we have diagnosed them since 2001, allowing up to 10 years of follow-up. These polyps are generally small and aren't a cause for concern.Ī fundic gland polyp with a diameter larger than about 2/5 inch (1 centimeter) carries a small risk of cancer. Fundic gland polyps are common among people who regularly take proton pump inhibitors to reduce stomach acid. Regular use of certain stomach medications. Familial adenomatous polyposis also can cause adenomas. When associated with this syndrome, fundic gland polyps are removed because they can become cancerous. This rare, inherited syndrome causes certain cells on the stomach's inner lining to form a type of polyp called fundic gland polyp. For that reason, they are generally removed. Hyperplastic polyps are unlikely to become cancerous, although those larger than about 2/5 inch (1 centimeter) carry a greater risk.Īdenomas are the least common type of stomach polyp but the type most likely to become cancerous. Also known as gastritis, this condition can cause the formation of hyperplastic polyps and adenomas. The most common causes of stomach polyps are: Stomach polyps form in response to damage to your stomach lining.
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